Endocrine, Metabolism & Exercise
2011a(14)97b(4) Describe the physiological effects of general anaesthesia on temperature regulation
2009b(11)00b(7)98a(7) Describe the role of insulin in fat metabolism
2008b(10): Describe sepsis and describe the metabolic consequences of sepsis.
2006b(16): Describe the physiological consequences of acute hypoglycaemia
2006a(13): Describe the factors that influence metabolic rate
2003a(15): Describe the physiological actions of thyroid hormones
2001b(3): Describe the fuel sources used during early and sustained fasting in man
2001a(5)/1996b(2): Explain briefly the role of the skin in maintaining a normal body temperature
1996b(8) Briefly explain how an ‘O2 debt’ arises and how the body deals with it
MAKEUP: Adrenocorticoid hormone synthesis
MAKEUP: Briefly discuss functions of the liver
HI Amanda,
Seasons greetings! I have just been reading about the tyrosine kinase insulin receptor and in your answer to 2000b(7) you state that is is a GPCR. Most textbooks seem to state that the tyrosine kinase protein is activated directly by insulin. There may some down stream g-protein activity somewhere, but I don’t think this system is really a GPCR. Cheers, Graham
Graham Wesley
December 31, 2010 at 9:59 am
G’Day Graham! Hope Xmas & New year’s weren’t too burdened by study! Thanks again for the feedback..the appropriate changes have been made!
Amanda
primarysaqs
January 9, 2011 at 10:23 am
Hi Amanda! With reference to: 2009b(11)/2000b(7)/1998a(7): Outline the actions of insulin that affect fat metabolism. The last point you made in your answer (nr 4) states an INcrease in glucose 6 phosphatase. Is this correct? Should it not be a decrease? See Kerry Brandis pg 171. Thanks! Theo.
Theo
February 15, 2011 at 7:22 pm
Hi Theo Thanks for pointing out the typo – appropriate corrections have been made.
AD
primarysaqs
February 16, 2011 at 5:21 pm
Hi Amanda,
Amazing site – greatly appreciated.
Just a (pedantically small) point regarding 2001a(5)96b(2) on the skin’s role in thermoregulation…
You have attributed incr. sweat production in raised ambient temp to increased SNS/ACh activity. It’s probably not SNS-related, as the overall impact of the hypothalamus is to DOWNregulate SNS activity when T(ambient) > T(TNZ). Looks like the sweat production is probably locally mediated (increased ACh release/sensitivity) as a direct result of higher temp. (http://jap.physiology.org/content/100/5/1692.long)
Cheers, JP
Josh P
May 29, 2012 at 5:25 pm
Hehe…scratch that! It’s both. There IS selective upregulation of SNS activity to sweat glands, with downreg in other areas. Smart thing, that hypothalamus!! JP
Josh P
June 5, 2012 at 10:00 pm
Hi Amanda, there was a TPN question asked in 07, asked again in Feb this year, is there an answer for it on your site somewhere I can’t see?
cheers
Michael Hicks
June 26, 2012 at 11:19 pm
Hi Michael
It appears not. Perhaps someone may submit the answer for posting on the site…
AD
primarysaqs
June 27, 2012 at 10:03 pm
Hi Amanda
Great work
Question on calcium metabolism has 2errors
Vit d activate osteoblasts so don’t increase resorption of bone and calcitonin inhibit osteoclasts not osteoblasts
Thank you
Ahmad
Ahmad
August 31, 2012 at 11:44 am