Primary Examination SAQs

Study notes for the ANZCA Primary Examination SAQs

Endocrine, Metabolism & Exercise

with 9 comments

2011a(14)97b(4) Describe the physiological effects of general anaesthesia on temperature regulation

2009b(11)00b(7)98a(7) Describe the role of insulin in fat metabolism

2008b(15): Describe the changes that occur with ageing that effect O2 delivery to the tissues during moderate exercise

2008b(10): Describe sepsis and describe the metabolic consequences of sepsis.

2008a(10)/2003a(11)/1996a(6): Define thermoneutral zone. Briefly explain how the body regulates temp when the ambient temperature exceeds the termoneutral zone

2008a(9): The skin, the kidneys, and the carotid bodies are examples of where specific organ blood flow is far in excess of that organ’s metabolic requirements. For each example, explain what the physiological role of the high organ blood flow is, why this high flow is an advantage to the person and a brief description of the mechanisms involved.

2006b(16): Describe the physiological consequences of acute hypoglycaemia

2006b(14)/1996a(5): Compare and contrast the physiological effects of a 6hr fast of fluids and food with a 24hr fast in a healthy adult

2006a(13): Describe the factors that influence metabolic rate

2003a(15): Describe the physiological actions of thyroid hormones

2001b(3): Describe the fuel sources used during early and sustained fasting in man

2001a(5)/1996b(2): Explain briefly the role of the skin in maintaining a normal body temperature

1996b(8) Briefly explain how an ‘O2 debt’ arises and how the body deals with it

MAKEUP: Adrenocorticoid hormone synthesis

MAKEUP: Briefly discuss functions of the liver

MAKEUP: Calcium absorption and metabolism

MAKEUP: Thyroid Hormone Synthesis

Written by primarysaqs

December 31, 2009 at 11:38 am

9 Responses

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  1. HI Amanda,
    Seasons greetings! I have just been reading about the tyrosine kinase insulin receptor and in your answer to 2000b(7) you state that is is a GPCR. Most textbooks seem to state that the tyrosine kinase protein is activated directly by insulin. There may some down stream g-protein activity somewhere, but I don’t think this system is really a GPCR. Cheers, Graham

    Graham Wesley

    December 31, 2010 at 9:59 am

    • G’Day Graham! Hope Xmas & New year’s weren’t too burdened by study! Thanks again for the feedback..the appropriate changes have been made!



      January 9, 2011 at 10:23 am

  2. Hi Amanda! With reference to: 2009b(11)/2000b(7)/1998a(7): Outline the actions of insulin that affect fat metabolism. The last point you made in your answer (nr 4) states an INcrease in glucose 6 phosphatase. Is this correct? Should it not be a decrease? See Kerry Brandis pg 171. Thanks! Theo.


    February 15, 2011 at 7:22 pm

    • Hi Theo Thanks for pointing out the typo – appropriate corrections have been made.



      February 16, 2011 at 5:21 pm

  3. Hi Amanda,
    Amazing site – greatly appreciated.
    Just a (pedantically small) point regarding 2001a(5)96b(2) on the skin’s role in thermoregulation…

    You have attributed incr. sweat production in raised ambient temp to increased SNS/ACh activity. It’s probably not SNS-related, as the overall impact of the hypothalamus is to DOWNregulate SNS activity when T(ambient) > T(TNZ). Looks like the sweat production is probably locally mediated (increased ACh release/sensitivity) as a direct result of higher temp. (

    Cheers, JP

    Josh P

    May 29, 2012 at 5:25 pm

    • Hehe…scratch that! It’s both. There IS selective upregulation of SNS activity to sweat glands, with downreg in other areas. Smart thing, that hypothalamus!! JP

      Josh P

      June 5, 2012 at 10:00 pm

  4. Hi Amanda, there was a TPN question asked in 07, asked again in Feb this year, is there an answer for it on your site somewhere I can’t see?


    Michael Hicks

    June 26, 2012 at 11:19 pm

    • Hi Michael

      It appears not. Perhaps someone may submit the answer for posting on the site…



      June 27, 2012 at 10:03 pm

  5. Hi Amanda
    Great work
    Question on calcium metabolism has 2errors
    Vit d activate osteoblasts so don’t increase resorption of bone and calcitonin inhibit osteoclasts not osteoblasts

    Thank you


    August 31, 2012 at 11:44 am

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